To the Editor: Parkinson's disease (PD) is a neurodegenerative disease that is the most common disease after Alzheimer's disease among neurological diseases. Destruction of the substantia nigra pars compacta of basal ganglia (BG) is the cause of the disease. The exact cause of this destruction is not yet known.1 Gait disorder is one of the cardinal symptoms of PD. The gait disorder in PD patients includes slowed gait, shortened length of stride, decreased rhythm and cadence, increased time of double support in the stance phase, shuffling and festinating gait, decreased swing of the arms, and disturbed regulation of the stride length. Five-minute walking can exhibit the disturbances in patients as compared with normal persons.1,2 Researchers have focused from different perspectives on PD, some of which are: introducing animal models, introducing conceptual models, presenting cognitive symptoms, and finding novel treatments such as drugs and deep brain stimulation (DBS).3–5 As studies shown, we can understand two main points about the brain and PD: