Contrary to earlier reported cases,3–5 in which as much as 5 g of clozapine could induce only 48 hours of coma, our index case showed clear discordance with reference to expected duration of recovery and the relative number of hours of coma heralded by as low as 2 g of clozapine. Fortunately, unlike in agranulocytosis and myocarditis, clozapine-induced coma continued to show a benign course, with complete recovery, as seen in our case. The severe sedation of clozapine toxicity responded well to armodafinil 25 mg bid, signifying its role in the management of somnolence of clozapine overdose.5 Although research started almost a decade ago, the fatal dose of clozapine is not yet clearly established, unlike that of benzodiazepines, barbiturates, opioids, and tricyclic antidepressants; and, just to speculate, if the usage of clozapine as a means of suicide attempt continues, we wonder whether the demarcating fatal dose could become more clear in the future. To date, no specific antidote exists for clozapine or, for that matter, any other antipsychotic toxicity, and forced diuresis and even techniques of extra-renal depuration are not effective because of the largely hepatic route of metabolism. Management of such patients with supportive care, proper monitoring, and armodafinil still holds the key to the successful outcome.